Click a link above to display the answer.











Lateral lobes.









Vermis.









Primary fissure.









Nodulus, floculus, and uvula.









Fastigial, Globose, Emboliform, and Dentate.









Dentate.









Globose and Emboliform.









Vermis, intermediate zone, and lateral zone.









Vermis.









Lateral zone.









Paravermal.









Pontocerebellum.









Vermis and intermediate zone.









controls movement and coordination of the trunk and proximal limb joints.









Vermis and flocculonodular lobe.









Motor planning for the extremeties.









Lateral corticospinal tract.









Distal limb coordination.









Rubrospinal and lateral corticospinal tracts.









Proximal limb and trunk coordination.









Tectospinal, reticulospinal, lateral vestibulospinal and anterior corticospinal tracts.









Balance and vestibule-ocular reflexes.









MLF, medial and lateral vestibulospinal tracts.









Output from deep cerebellar nuclei (primarily) and inputs from ventral spinocerebellar tract.









Inputs from pontine nuclei.









Inputs from inferior olive, vestibular nuclei, dorsal spinocerebellar tract, and accessory cuneate nucleus.









Superior.









Inferior.









Middle.









Primarily axons of granule cells and dendrites of purkinje cells.









Granule cell.









Purkinje, basket, stellate, and golgi cells.









Mostly excitatory (but some inhibitory).









Dentate.









Emboliform and globose (interposed).









Fastigial









Vestibular nuclei in brainstem.









Output is carried exclusively by purkinje cells.









They inhibit deep cerebellar and vestibular nuclei.









Exclusively from the inferior olivary nucleus.









They excite deep cerebellar nuclei and purkinje cells.









Climbing and mossy fibers.









One.









Around ten.









All sites of cerebellar cortical input except the inferior olivary nuclei.









They excite deep cerebellar nuclei and granule cells.









In the granular cell layer of the cerebellar cortex.









They send their axons up into the molecular layer and excite an entire folium of purkinje cells, stellate cells, and basket cells.









They form inhibitory synapses on the dendrites of multiple adjacent purkinje cells in a direction perpendicular to the folia.









In the molecular layer of the cerebellar cortex.









They form inhibitory synapses on the soma of multiple adjacent purkinje cells in a direction perpendicular to the folia.









In the granular cell layer of the cerebellar cortex.









They form an inhibitory feedback system with granule cells (they inhibit granule cells upon being excited by them).









Purkinje cells of the flocculonodular lobe synapse on cells of the vestibular nuclei.









Excitatory from inferior olive and vestibular sensory receptors (ear); Inhibitory from purkinje cells of flocculonodular lobe.









Vestibular nuclei









Mossy fibers to the granule cells of the flocculonodular lobe.









Contralatera?









Loss of equilibrium (swaying and ataxia), nystagmus, and nausea; No tremors.









Excitatory from inferior olivary nucleus, dorsal and ventral spinocerebellar tracts, and cuneocerebellar tract.









Chronic alcoholism and familial cerebellar degeneration.









Ataxia of legs with tendency to fall toward side of lesion; Staggering wide-based gait.









Anterior lobe.









Contralateral superior cerebellar peduncle.









Red nucleus, VA, and VL.









Titubations (tremor of trunk, resembles little worms) and ataxia.









Climbing fibers from the inferior olive to the dentate nucleus and purkinje cells; Mossy fibers to contralateral dentate and granule cells.









Lateral cerebellum => red nucleus =(CTT)=> inferior olivary nucleus =(mossy fibers)=> lateral cerebellum.









Lateral cerebellum => VA and VL => motor cortex => pontine nuclei => lateral cerebellum.









It allows the cortex to have limited control of cerebellar functions.









Hypotonia, ataxia, dysmetria, dysdiadochokinesis, tremor, and speech disturbances.









Overstimulation of vestibular nuclei leads to temporary dysfunction of prkinje cells due to glutamate excitotoxicity.